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These are electrolytes that help increase hydration in the body and help to maintain body fluid balance

Excitotoxicity, calcium overload and membrane permeability transition, metabolic and bioenergetic failure, antioxidants depletion, free radicals over production and oxidative stress, elevated calpains, caspases, and apoptosis inducing factors are key mechanisms governing mitochondria-mediated neuronal damage following TBI [18, 19]

oryzae 007 infection, cytoplasmic Ca 2+ influx began to appear at 12 hpi, increased markedly at 24 hpi, and peaked at 36 hpi ( Figures 2A, C )

The interplay between oxidative stress and brain-derived neurotrophic factor modulates the outcome of a saturated fat diet on synaptic plasticity and cognition

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