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At the same time, immune cells impose reciprocal metabolic constraints on cancer stem-like cells through oxidative stress, nutrient restriction, and inflammatory metabolites, which drive cancer stem-like cells plasticity, epithelial-to-mesenchymal transition (EMT)-like programs, and epigenetic remodeling

A main emerging concept is that these two neural cell types have different and energetically adapted glyoxalase defense mechanisms which may serve as protective mechanism against methylglyoxal-induced cellular damage

Briefly, HCT116 p53+/+ cells (4 10 4 per well) were coated in a 12-well plate

2 nd was supported by the 1R25CA240137 UPWARDS Training Program and the CPRIT Research Training Award CPRIT Training Program (RP210028) was omitted

coli BL21 cells and confirmed by sequencing