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Chronic hyperglycemia in DM disrupts normal mitochondrial function leading to excessive ROS generation, advanced glycation end-product (AGE) formation, and activation of pro-inflammatory pathways [29]

[] Furthermore, BNIP3, a critical mitophagy receptor, is a known transcriptional target of FOXO3, which itself can stimulate PGC-1 expression

severe impairment, n = 4)

The results showed that the vast majority of apoptotic cells were neuronal cells, suggesting that sulindac exerts adverse effects on early neurodevelopment in zebrafish larvae

The binding between the nickel ion and the agarose support is so strong that it will not be disrupted by high concentration of many commonly used chemicals (see Table 1 for tested chemicals)