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Pharmacologic activation of mitochondrial biogenesis exerts widespread beneficial effects in a transgenic mouse model of Huntingtons disease

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4-HNE can form adducts with Fas, a death receptor protein with a cysteine-rich extracellular domain (81)

However, its powerful effect on GH means you have to watch for the usual GH-related side effects like fluid retention, achy joints, or injection site reactions