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In the canonical Hippo pathway, Mst1/2 kinases form a complex with SAV1 to activate Lats1/2 kinase, which subsequently phosphorylate and inhibit the transcriptional co-activators YAP and TAZ

141,142,143 Both Dengue virus and Zika virus exploit the receptor tyrosine kinase AXL as an entry mediator for host infection, whereas the epidermal growth factor receptor functions as a coreceptor for adeno-associated virus serotype 6 and human cytomegalovirus to facilitate their entry into the host

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