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[255-258] We conclude that NO (nitric oxide) plays a critical immunoregulatory role and NO modulation may prevent the onset of autoimmune reactions[259] Research has shown that IL-17 and the TH-17 system activates iNOS and initiates tissue destruction in autoimmunity: Interleukin-17 is a proinflammatory cytokine involved in autoimmune disorders

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Altogether, these findings indicate that the TRPM2-mediated calcium influx pathway in microglia, particularly enhanced in reactive cells, has opened new research opportunities, suggesting that modulating TRPM2 activity could represent a strategy to regulate microglial function