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G., Neumann, J

Here, we summarized the role of the vicious cycle of mitochondrial damage and ROS accumulation in RA and introduced potential drugs that target mitochondria or scavenge ROS for RA treatment, with the aim of providing some help for the clinical treatment of RA ( Table 1 )

[124], these decreases are frequently accompanied by increased levels of malondialdehyde (MDA) and total oxidant status (TOS), which indicate continued oxidative intake of antioxidants

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Conclusion: Hepatocyte II-spectrin knockdown decreased PKM2 expression in Kupffer cells, suppressing pro-inflammatory cytokine production, and blocking NASH-associated HCC