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Across tumors, DOX resistance commonly engages stress-induced autophagy programs, classically AMPK/ULK1 activation with mTOR suppression which sustain survival by recycling substrates and buffering ROS

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To provide different perspectives on the estimated treatment effect in weight management RCTs, we recommend that future publications investigating new or already-approved weight-loss medications report the treatment policy estimand and the trial product estimand

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