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Therefore, here we used hyperphenylalaninemic (Hph-1) mice, deficient in GTPCH with the heterozygotes and homozygote animals producing different BH 4 levels to demonstrate the relationship between BH 4 and NOS output, and sepiapterin supplementation to investigate the impact of an improved BH 4 supply in the ischaemic phase of the murine OIR model

And nobody connected the dots because skin and joints are treated as separate departments in a system that was not designed to see the whole person

3A) and the downregulation of EBNA1 was verified (Fig