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Further, in a mouse model of Alzheimer's disease, NMN was found to reduce the levels of amyloid precursor protein (APP), reverse the oxygen consumption deficit in brain mitochondria, and reduce mitochondrial fragmentation in neurons, thereby improving the mitochondrial respiratory defect [40]

S1P is a bioactive sphingolipid molecule that regulates various cellular functions, including cell survival, differentiation, migration, proliferation, and immune responses, through its binding to five G protein-coupled receptors (S1PR1 to S1PR5) [69]

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Here, the inhibition of wounding-induced ROS accumulation significantly inhibited healing in planarian worms by preventing F-actin reorganisation and epithelial cell rearrangements mediated through c-JUN activation at the wound site 122